Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Alternatively, our capability to rescue SERT perform in KI midbrain synaptoneurosomes via the inhibition of FAK suggests elevated FAK signaling downstream in the Pro32Pro33 mutant, as confirmed by amplified pFAK localization in five-HT synapses. https://manuelsgsen.blog-ezine.com/32371212/everything-about-pro33